Lipitor (Atorvastatin)

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Lipitor (Atorvastatin)
BEAT HEART DISEASE WITHOUT SURGERY: EXAMINING THE EFFECTS-HOW EDTA WORKS AND THE CALCIUM FACTOR
The action of EDTA, like the disease it treats, is multifarious. It seems to have a regulatory action on many vital systems of the body, systems which tend to malfunction with increasing age.
Whether this is because it clears body systems of unwanted debris, thus allowing them to get on with their work, or whether its action is one of chemical intervention, is really irrelevant to the atheroma sufferer. However from the scientific point of view, the main benefits seem to fall under several main categories: removal of calcium, regulation of the clotting factor, stimulation of enzyme production, and last but by no means least,” removal of poisons and system suppressives such as heavy metals.
The Calcium Factor-Calcium is one of the commonest minerals in the world. From earliest years we in the west are brought up with a large amount of it in our diets. Instead of beginning with the moderately calcified breast milk and being weaned onto a variety of solids, we are brought up on cow’s milk, which was designed for an animal with bones many times larger and denser than ours.
Dairy produce has become a western favourite and many ‘treat’ foods are composed around it – ice cream, cream itself, cream cheese, fromage frais (give a product a trendy name and sell it all over again), milk shakes, yoghurt drinks and yoghurt itself, cheese in its many forms, etc.
There is also abundant calcium in many of the popular foods we eat, besides it being in drinking water. But for now let’s assume that it is one mineral of which we are not in short supply.
Whether due to excesses in the diet or not, something seems to go wrong with the calcium metabolism around middle age. It is a metabolism which is constantly in and out of play in the body, since blood levels of it have to be kept within a critically narrow band, and to achieve this, thyroid and parathyroid hormones work in balanced opposition to each other, secreting hormones (calcitonin and parahormone) which stimulate the release of calcium from body stores, such as bones, or suppressing this activity by releasing hormones which stimulate calcium to be laid down in bone.
Calcium therefore moves from easily transportable states which dissolve in blood to
non-soluble states which can then be stored. It is this transportable ionic calcium which largely becomes part of arterial plaque and incidentally can become part of other calcified deposits in the body such as those around joints and old injuries. It is then known as metastatic calcium. (NB: ions are atoms or groups of atoms which carry an electrical charge, either positive or negative, of which more later.)
The minerals of the body work in harmony with each other but also in juxtaposition – so that one is balanced against another to achieve metabolic results. If the diet constantly provides excesses of one and deficiencies of another, then mineral imbalances occur and the body somehow has to cope with this by finding storage places for the excess and attempting to overcome problems caused by the deficiencies.
This is what is thought to happen when the body starts laying down calcium on artery walls. There is another hypothesis which suggests that this happens because the body ‘perceives’ that the arterial walls are becoming injured (by the action of destructive free radicals) and hence it deploys calcium as it does in other injury sites, to shore up the damage. Whatever the reason, the result is an accumulation of deposits which block circulation and prevent reabsorption of other plaque components that get shored up as well, such as fats, blood cells (especially platelets which are also used for repair purposes) and fibrous material, such as elastin and collagen.
Cardio & Blood Bruce Halstead writes in The Scientific Basis of EDTA Chelation Therapy that ‘there is an increasing amount of evidence that an abnormality in calcium metabolism is the biochemical lesion primarily responsible for heart failure’. He goes on to say that the lesion is believed to be caused by a difficulty arising in the exchange of the action potential involving calcium ions due to problems in the conducting cell membranes.
What this all amounts to in lay terms is that heart attacks can and do occur because of a faulty calcium metabolism on two levels: firstly, on the accumulative level of progressive calcification of arteries, including coronary arteries, and secondly, on the muscle spasm level due to the malfunction of heart cells themselves. Keeping in mind that mental and emotional input also affect this most sensitive organ and you may have the situation of a heart attack occurring where there is no significant blockage of the coronary arteries. All the more reason to get to grips with the causes, be they metabolic and therefore probably dietary or stress-related. But there are one or two other factors in this complex equation worth mentioning.
*45/104/2*

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