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BEAT HEART DISEASE WITHOUT SURGERY: EXAMINING THE EFFECTS-THE ENZYME FACTOR
Enzymes play a vital part in maintaining the integrity of the arterial walls. Enzymes are organic catalysts which enable chemical processes to take place at body temperatures. These enzyme activities are quite efficient in youth but, according to Morton Walker and others, ’starting in the fourth decade of life, they begin to diminish in supply to about one-half of their previous activity’.
He goes on to describe how this reduction decreases the repair process of arteries which in turn leads to overcompensatory activities which encourage the aggregation of cholesterol and calcium to form plaque. This in turn attracts platelets which collide with the plaque and in response to the perception of injury, release their own hormones, which in this case are likely to be the ones which prompt coagulation.
Bruce Halstead explains that enzyme activity depends on metal ions for its completion and any distortion in the balance of these is once again likely to promote degenerative activity in the cells. The balance, he says between calcium and magnesium, if distorted (back to dietary imbalances again) leads to excessive calcium concentration which leads in turn to further inhibition of enzymes. This may result in damage to cell function, respiration, and produce an oxygen deficiency state (anoxia) all of which is conducive to chronic degenerative disease.
He also notes the resultant reduction in enzyme activity in artery walls: ‘Enzymatic analyses have shown (Kirk 1959, 1968) that of the 98 enzyme systems in the artery wall, 46 of them are in a state of depressed activity in arteriosclerotic tissue.’
Cardio & Blood He goes on to say, ‘These are important biochemical events in atherogenesis and provide important target sites for the action of EDTA therapy in the removal of calcium and other transitional metals, and aiding in the normalization of enzymatic homeostasis.’
This would seem to add weight to the opinion expressed by Fritz Schellander and others that a prime action of EDTA is removing unwanted metals so that regeneration can take place. Schellander goes further and suggests that the excessive presence of calcium in arterial plaque is in fact part of the body’s attempt to heal: ‘The protective function of calcium laid down around tuberculous lesions or inflamed tendons is well-accepted. Could there be a similar mechanism operating in our arteries?’
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