Atropisol (Atropine)

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Atropisol (Atropine)
MULTIPLE SCLEROSIS: WHY EVENING PRIMROSE OIL IS BETTER THAN LINOLEIC ACID
The results of the 1978 Newcastle study and the 1973 London and Belfast study might lead one to believe that sunflower seed oil, sunflower seed oil spreads and similar sources of linoleic acid were the most effective PUFAs for MS.
It is indeed a very good idea to use such seed oil products in one’s daily diet. However, there are still very good reasons for supplementing the diet with evening primrose oil.
The most important reason is that, no matter how much linoleic acid you consume, there could be some problems metabolizing it. Unless the linoleic acid is converted inside the body to longer-chain fatty acids and prostaglandins, it is of little use in MS.
Many things can get in the way of converting all the linoleic acid you eat. These are the blocking agents. The most common ones are:
Ђў Foods rich in saturated fat
Ђў Foods rich in cholesterol
Ђў Foods rich in trans fatty acids
Ђў Stress, when the body releases catecholamine
Ђў A diet too high in simple sugars
Ђў Low levels of zinc, magnesium, Vitamin C, Vitamin B6, nicotinamide (B3), and biotin
Ђў Alcohol in moderate to large amounts
Ђў Viral infections
Ђў Atopic conditions
There are very few people with MS who do not have several of these ‘blocking agents’ operating in their own lives, before they start any kind of self-help therapy.
The virtue of evening primrose oil is that it begins later on in the metabolic pathway, completely avoiding most of the blocking agents which for the most part inhibit the first step – the conversion of linoleic acid to gammalinolenic acid. Evening primrose oil, unlike sunflower seed oil, is rich in gammalinolenic acid, thus starting at step 2 in the metabolic pathway. So when you take evening primrose oil, you can be sure it will convert into the longer-chain fatty acids, and prostaglandins. With sunflower seed oil, you cannot be sure of this at all.
*30/60/5*

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